Скачать или смотреть Pathogenesis or Mechanism Of Action Cell Injury in Pathophysiology | B. Pharma Semester 2(Lecture 2)

Pathogenesis or Mechanism Of Action Cell Injury in Pathophysiology | B. Pharma Semester 2(Lecture 2)

इस वीडियो में हम Cell Injury के Pathogenesis को सरल भाषा में समझेंगे। B. Pharma 2nd semester के students के लिए यह वीडियो Pathophysiology की महत्वपूर्ण topic को cover करता है।
इस वीडियो में आप जानेंगे:
Cell injury kya hota hai
Pathogenesis ke mechanisms
Causes of cell injury
Types of cell injury (Reversible & Irreversible)
Examples for easy understanding
यह वीडियो आपके exam preparation और concept clear करने के लिए perfect है।
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your Queries

Cell injury
Pathogenesis
Causes of cell injury
Hypoxia
Ischemia
Free radicals
Oxidative stress
Chemical injury
Physical injury
Infectious agents
Genetic defects
Nutritional imbalance
Apoptosis
Necrosis
Cellular adaptation
Atrophy
Hypertrophy
Hyperplasia
Metaplasia
Dysplasia
Reversible injury
Irreversible injury
Mitochondrial damage
Membrane damage
Calcium influx
Protein misfolding
Lipid peroxidation
Inflammation
Cell death mechanismsReactive oxygen species
Mitochondrial dysfunction
Endoplasmic reticulum stress
DNA damage
Protein denaturation
Lipid peroxidation
Inflammatory mediators
Cytokines
Cellular swelling
Vacuolation
Blebbing
Karyolysis
Pyknosis
Karyorrhexis
Ischemia-reperfusion injury
Toxic injury
Heavy metal toxicity
Drug-induced cell injury
Radiation injury
Free radical scavengers
Antioxidants
Necroptosis
Ferroptosis
Autophagy
Cellular stress responseOxidative phosphorylation defect
ATP depletion
Membrane permeability
Lysosomal rupture
Enzyme release
Free radical injury
Superoxide anion
Hydrogen peroxide
Hydroxyl radical
Nitric oxide
Protein cross-linking
Cytoskeletal damage
Lipid accumulation
Glycogen depletion
Cellular hypoxia
Anaerobic metabolism
Lactic acidosis
Inflammation-induced injury
Immune-mediated cell damage
Viral cytopathic effect
Bacterial toxins
Endotoxins
Exotoxins
Oxidative DNA damage
Redox imbalanceEndothelial injury
Vascular permeability
Cell signaling pathways
Caspases
Bcl-2 family proteins
Mitochondrial permeability transition
Oxidative stress markers
Lipid peroxidation products
Malondialdehyde
Glutathione depletion
Superoxide dismutase
Catalase
Necrosis types (coagulative, liquefactive, caseous)
Apoptotic bodies
Phagocytosis of dead cells
Inflammatory cell infiltration
Neutrophil-mediated injury
Macrophage activation
TNF-alpha
IL-1, IL-6
Chemokines
Hypoxia-inducible factor
Reactive nitrogen species
Peroxynitrite
Stress granulesMitochondrial ROS
Calcium overload
Membrane blebbing
Nuclear shrinkage
Chromatin condensation
Cell lysis
Endoplasmic reticulum calcium release
Protein aggregation
Ubiquitin-proteasome pathway
Autophagosome formation
Mitophagy
Cellular senescence
Stress-activated protein kinases
JNK pathway
p38 MAPK pathway
Oxidative DNA strand breaks
Lipid droplet accumulation
Cytokine storm
Redox signaling
Mitochondrial swelling
Phosphatidylserine exposure
Immune-mediated apoptosis
Complement-mediated cell injury
Hypoxic injury
Reperfusion injuryOxidative mitochondrial injury
Cytochrome c release
Apoptosome formation
DNA fragmentation
PARP activation
Caspase cascade
Cell shrinkage
Chromatin margination
Necroptosis signaling
Ferroptotic cell death


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