Cell type-specific vulnerability to traumatic brain injury

Presented By: Araks Martirosyan, Ph.D.

Speaker Biography: Dr. Araks Martirosyan is a sr. postdoctoral researcher in the Laboratory of Glia Biology at the VIB-KU Leuven Center for Brain and Disease Research (Belgium). After Finishing her B.Sc. in the Faculty of Physics at Yerevan State University (Armenia) and M.Sc. in Applied Physics at Cergy-Pontoise University (France), she became interested in the application of Statistical Physics to Biology and chose Computational Biology as her future research direction. Dr. Martirosyan received her Ph.D. from the Sapienza University of Rome (Italy) in 2016.

Webinar: Cell type-specific vulnerability to traumatic brain injury

Webinar Abstract: Traumatic brain injury (TBI) is best characterized as brain dysfunction caused by an outside force, usually a violent blow to the head, often occurring as a result of a severe sports injury or car accident. It is a major global health concern, affecting 69 million people worldwide per year, and is one of the most common causes of disability and death in adults. Nearly 50% of TBI patients experience long term cognitive impairment, linked to hippocampal neuronal damage and death. At the same time a deregulation of the hippocampal neurogenic processes is observed which impedes possible functional recovery of the brain. In particular, immunohistochemistry in mouse hippocampus has shown that cortical TBI affects neural stem and progenitor cells (NSPCs) residing in sub-granular zone of dendrite gyrus.

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